Vasculata 2018 St. Louis, MO July 23 - 26, 2018
Supported in part by the:
Vascular Biology
Newport, RI
October 14-18, 2018
Lymphatic Forum 2019 Austin, TX May 31 - June 1, 2019
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Please review our policy as time permits so you have a complete understanding of the data we have, why we have it, and how we use it.
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Vascular Biology 2018 - October 14-18 in Newport!
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New Venue for Vascular Biology
Vascular Biology 2018 will feature the
Vascular Inflammation Workshop and the
Biology of Signaling in the Cardiovascular System Workshop. Our new venue, in Newport, RI, is Gurney's Newport Resort and Marina located on Goat Island. Just a 10 minute walk from downtown Newport!
Plan now to attend! Go to
www.navbo.org/vb2018
for the meeting program, registration, abstract submission, award applications and more!
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Attend Vasculata 2018 in St. Louis
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Plan to attend Vasculata this summer July 23-26 in St. Louis
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Sign up now for our August 2 Webinar with Michael Dellinger
Our webinar attendees requested a webinar on Lymphatics, so please join us on August 2 at 1:00pm EDT for Dr. Dellinger's presentation,
"Lymphatic Vessels and Vanishing Bones: Animal Models of Lymphatic Anomalies with Bone Involvement." Dr. Dellinger is an Assistant Professor at UT Southwestern Medical Center.
The webinar is sponsored by the Lymphatic Malformation Institute and the Lymphangomatosis & Gorham's Disease Alliance.
To register for the webinar please go to
http://www.navbo.org/events/webinars/718-webinar082018
Please note: we will not be holding a July webinar
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Registration for Vasculata closes on July 20
Early bird registration ends:
Vascular Biology 2018 - August 15
Abstract Submission Deadline:
Vascular Biology 2018 - August 1
Nominate a colleague for a Meritorious Award:
Nominations for the 2019 Earl P. Benditt Award and the Judah Folkman Award in Vascular Biology are due July 8.
NAVBO Travel Awards are available for the following meetings:
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Bioinformatics at Vascular Biology 2018!!!
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Watch the webinar and see how bioinformatics can enhance your research
(click on the slide to start the movie)
Attend Vascular Biology 2018 and register for this special session!
Bring your lap top and you favorite gene!!
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Applications solicited for Science and SciLifeLab's Prize for Young Scientists
For the sixth year, Science/AAAS and SciLifeLab, a coordinated effort of four universities, have joined forces to recognize outstanding recent doctoral graduates in the life sciences (Ph.D. awarded between January 1, 2016, and December 31, 2017). This award, intended to provide encouragement to young scientists beginning their scientific careers, includes a grand prize of $30,000, as well as $10,000 to winners in each of three other subject categories (Cell and Molecular Biology; Genomics and Proteomics; Ecology and Environment; Translational Medicine).
Applications are due July 15, 2018. Winners will have their essays prominently published and will be honored in Stockholm, Sweden, in December of this year.
(from the July 14 issue)
published recently in The Wall Street Journal. Lublin, by now a veteran editor for the WSJ, has some hard-earned pointers on negotiating numerous workplace issues, including unbridled gender bias and unevenly applied corporate policies. Although Lublin has pursued a profession somewhat afield from medical research, there is an abundance of wisdom in her reflections that is well worth the attention of eager trainees approaching their first independent positions.
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The Lab of Dr. C. Keith Ozaki
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Welcome to our New Members:
Timothy Sargis, University of Illinois at Chicago
Scott Wong, Boston Children's Hospital
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Recent Publications by NAVBO Members
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CD301b/MGL2+ Mononuclear Phagocytes Orchestrate Autoimmune Cardiac Valve Inflammation and Fibrosis Circulation Background: Valvular heart disease is common and affects the mitral valve (MV) most frequently. Despite the prevalence of MV disease (MVD), the cellular and molecular pathways that initiate and perpetuate it are not well understood. Read more Redox Regulation of Mitochondrial Fission Protein Drp1 by Protein Disulfide Isomerase Limits Endothelial Senescence Cell Reports Mitochondrial dynamics are tightly controlled by fusion and fission, and their dysregulation and excess reactive oxygen species (ROS) contribute to endothelial cell (EC) dysfunction. Read more Deubiquitinase function of A20 maintains and repairs endothelial barrier after lung vascular injury Cell Death Discovery Vascular endothelial cadherin (VE-cad) expression at endothelial adherens junctions (AJs) regulates vascular homeostasis. Read more Clinical factors that influence the cellular responses of saphenous veins used for arterial bypass Journal of Vascular Surgery OBJECTIVE: When an autogenous vein is harvested and used for arterial bypass, it suffers physical and biologic injuries that may set in motion the cellular processes that lead to wall thickening, fibrosis, stenosis, and ultimately graft failure. Read more Determining the Pathogenicity of a Genomic Variant of Uncertain Significance Using CRISPR/Cas9 and Human-Induced Pluripotent Stem Cells Circulation Background -The progression toward low-cost and rapid next-generation sequencing has uncovered a multitude of variants of uncertain significance (VUS) in both patients and asymptomatic "healthy" individuals. Read more Progress, obstacles, and limitations in the use of stem cells in organ-on-a-chip models Advanced Drug Delivery Reviews In recent years, drug development costs have soared, primarily due to the failure of preclinical animal and cell culture models, which do not directly translate to human physiology. Read more EMAPII Monoclonal Antibody Ameliorates Influenza A Virus-Induced Lung Injury Molecular Therapy
Influenza A virus (IAV) remains a major worldwide health threat, especially to high-risk populations, including the young and elderly. There is an unmet clinical need for therapy that will protect the lungs from damage caused by lower respiratory infection. Here, the authors analyzed the role of EMAPII, a stress- and virus-induced pro-inflammatory and pro-apoptotic factor, in IAV-induced lung injury. Read more
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Industry News (from June 14 issue)
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New hypertension guidelines explored in leading AHA journal
Task force calls for increased inclusion of pregnant and lactating women in clinical studies A task force, charged by the US Department of Health and Human Services with identifying gaps in research on pregnant or lactating women, has finalized its work, calling for increased inclusion and integration of women from these groups into clinical trials. As reported by MedPage Today, the study collective, named PRGLAC (Research Specific to Pregnant Women and Lactating Women), approved 15 recommendations, including the removal of pregnant women as a vulnerable population in the common rule, and the inclusion of pregnant women and lactating women to capture and report maternal, obstetric, and neonatal outcomes, including pregnancy complications such as pre-eclampsia. Panelists from multiple government agencies (NIH, NICHD, FDA, and the National Vaccine Program Office) as well as members of industry and various professional organizations participated on the task force's efforts.
Annoyance in response to noise linked to dangerous cardiovascular ourcomes
A
new report published in the International Journal of Cardiology explores the relationship between environmental noise and the genesis of cardiac arrhythmias. Already studied in vascular disease, the new findings link responses to experiencing extreme aural stimulation (e.g., noise from aircraft, road, rail, and one's neighborhood) to increased incidence of atrial fibrillation. Atrial fibrillation, the most common type of arrhythmia, can lead to thrombosis, stroke, and heart failure. Nearly three million Americans are affected by atrial fibrillation. Noise annoyance, defined as noise-provoked responses such as anger, sleep disruption and subsequent exhaustion, and stress. Study participants distinguished between noise annoyances during the day and at night and recorded their responses using standardized questionnaires. Mechanisms connecting the noise annoyance responses and cardiac dysfunction remain to be defined.
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